Many individuals with autism spectrum disorder (ASD) face challenges in social interactions, which constitutes a significant societal concern. Recently, the chromatin remodeling factor CHD8 has been identified as the most promising candidate gene for ASD. We have generated mice that replicate the CHD8 mutation observed in individuals with ASD and demonstrated that these mice exhibit ASD-like symptoms due to delayed neurogenesis and abnormalities in neural circuits. In this study, we aim to identify cell populations involved in the development of ASD during neurogenesis and to characterize the neural circuits constructed by these cells, with the ultimate goal of developing evidence-based treatments.